Autophagy Gives a Nod and a Wink to the Inflammasome and Paneth Cells in Crohn's Disease
- 1 November 2008
- journal article
- preview
- Published by Elsevier in Developmental Cell
- Vol. 15 (5) , 641-642
- https://doi.org/10.1016/j.devcel.2008.10.009
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- Impaired Autophagy of an Intracellular Pathogen Induced by a Crohn's Disease Associated ATG16L1 VariantPLOS ONE, 2008
- Loss of the autophagy protein Atg16L1 enhances endotoxin-induced IL-1β productionNature, 2008
- A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cellsNature, 2008
- The Atg16L Complex Specifies the Site of LC3 Lipidation for Membrane Biogenesis in AutophagyMolecular Biology of the Cell, 2008
- Mechanism of IL-1β-Induced Increase in Intestinal Epithelial Tight Junction PermeabilityThe Journal of Immunology, 2008
- Toll-like receptors control autophagyThe EMBO Journal, 2008
- Unveiling the roles of autophagy in innate and adaptive immunityNature Reviews Immunology, 2007
- Intracellular NOD-like Receptors in Host Defense and DiseaseImmunity, 2007
- Unravelling the pathogenesis of inflammatory bowel diseaseNature, 2007
- Human IRGM Induces Autophagy to Eliminate Intracellular MycobacteriaScience, 2006