TGF-β/Smad signaling inhibits IFN-γ and TNF-α-induced TARC (CCL17) production in HaCaT cells
- 1 February 2003
- journal article
- research article
- Published by Elsevier in Journal of Dermatological Science
- Vol. 31 (1) , 53-58
- https://doi.org/10.1016/s0923-1811(02)00141-x
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 17 references indexed in Scilit:
- Transforming growth factor‐bβ1 suppresses atopic dermatitis‐like skin lesions in NC/Nga miceClinical and Experimental Allergy, 2002
- TARC: novel mediator of allergic inflammationClinical and Experimental Allergy, 2001
- A Th2 Chemokine, TARC, Produced by Keratinocytes May Recruit CLA+CCR4+ Lymphocytes into Lesional Atopic Dermatitis SkinJournal of Investigative Dermatology, 2000
- Roles of Bone Morphogenetic Protein Type I Receptors and Smad Proteins in Osteoblast and Chondroblast DifferentiationMolecular Biology of the Cell, 1999
- Overproduction of Th2-specific chemokines in NC/Nga mice exhibiting atopic dermatitis–like lesionsJournal of Clinical Investigation, 1999
- The chemokine receptor CCR4 in vascular recognition by cutaneous but not intestinal memory T cellsNature, 1999
- Selective recruitment of CCR4-bearing Th2 cells toward antigen-presenting cells by the CC chemokines thymus and activation-regulated chemokine and macrophage-derived chemokineInternational Immunology, 1999
- TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4The EMBO Journal, 1997
- The T Cell-directed CC Chemokine TARC Is a Highly Specific Biological Ligand for CC Chemokine Receptor 4Journal of Biological Chemistry, 1997
- Stimulation of erythropoiesis by in vivo gene therapy: physiologic consequences of transfer of the human erythropoietin gene to experimental animals using an adenovirus vectorBlood, 1994