Upregulation of Vascular NAD(P)H Oxidase Subunit gp91phox and Impairment of the Nitric Oxide Signal Transduction Pathway in Hypertension
- 3 August 2001
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 285 (5) , 1130-1135
- https://doi.org/10.1006/bbrc.2001.5312
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- A gp91phox Containing NADPH Oxidase Selectively Expressed in Endothelial Cells Is a Major Source of Oxygen Radical Generation in the Arterial WallCirculation Research, 2000
- NAD(P)H OxidaseCirculation Research, 2000
- Endothelin-1 Induces NAD(P)H Oxidase in Human Endothelial CellsBiochemical and Biophysical Research Communications, 2000
- Endothelial Function in HypertensionHypertension, 1999
- Cell transformation by the superoxide-generating oxidase Mox1Nature, 1999
- Interactions between NO and reactive oxygen species: pathophysiological importance in atherosclerosis, hypertension, diabetes and heart failureCardiovascular Research, 1999
- Increased NAD(P)H oxidase-mediated superoxide production in renovascular hypertension: Evidence for an involvement of protein kinase CKidney International, 1999
- p22phox mRNA Expression and NADPH Oxidase Activity Are Increased in Aortas From Hypertensive RatsCirculation Research, 1997
- Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone.Journal of Clinical Investigation, 1996
- Abnormal Endothelium-Dependent Vascular Relaxation in Patients with Essential HypertensionNew England Journal of Medicine, 1990