Suprabasal integrin expression in the epidermis of transgenic mice results in developmental defects and a phenotype resembling psoriasis
- 1 December 1995
- Vol. 83 (6) , 957-968
- https://doi.org/10.1016/0092-8674(95)90211-2
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Response of psoriasis to a lymphocyte-selective toxin (DAB389IL-2) suggests a primary immune, but not keratinocyte, pathogenic basisNature Medicine, 1995
- Integrin Expression by Human Epidermal Keratinocytes Can Be Modulated by Interferon-γ, Transforming Growth Factor-β, Tumor Necrosis Factor-α, and Culture on a Dermal EquivalentJournal of Investigative Dermatology, 1995
- Integrin αvβ3 antagonists promote tumor regression by inducing apoptosis of angiogenic blood vesselsCell, 1994
- Functional down-regulation of α5β1 integrin in keratinocytes is reversible but commitment to terminal differentiation is notJournal of Cell Science, 1993
- Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation.The Journal of cell biology, 1992
- Cachexia and graft-vs.-host-disease-type skin changes in keratin promoter-driven TNF alpha transgenic mice.Genes & Development, 1992
- Aberrant integrin expression during epidermal wound healing and in psoriatic epidermis.Journal of Clinical Investigation, 1992
- Spontaneous inflammatory disease in transgenic rats expressing HLA-B27 and human β2m: An animal model of HLA-B27-associated human disordersCell, 1990
- Elevated levels of the α5β1 fibronectin receptor suppress the transformed phenotype of Chinese hamster ovary cellsCell, 1990
- Limited diversity of γδ antigen receptor genes of thy-1+ dendritic epidermal cellsCell, 1988