Therapeutic potential of treating chronic obstructive pulmonary disease (COPD) by neutralising granulocyte macrophage-colony stimulating factor (GM-CSF)

This publication has 80 references indexed in Scilit:

• Innate immune responses to LPS in mouse lung are suppressed and reversed by neutralization of GM-CSF via repression of TLR-4
  American Journal of Physiology-Lung Cellular and Molecular Physiology, 2004
• Alveolar Macrophages as Orchestrators of COPD
  COPD: Journal of Chronic Obstructive Pulmonary Disease, 2004
• PU.1 regulation of human alveolar macrophage differentiation requires granulocyte-macrophage colony-stimulating factor
  American Journal of Physiology-Lung Cellular and Molecular Physiology, 2003
• Chronic obstructive pulmonary disease: molecular and cellularmechanisms
  European Respiratory Journal, 2003
• Endocytic Internalization of Adenovirus, Nonspecific Phagocytosis, and Cytoskeletal Organization Are Coordinately Regulated in Alveolar Macrophages by GM-CSF and PU.1
  The Journal of Immunology, 2002
• GM-CSF, via PU.1, regulates alveolar macrophage FcγR-mediated phagocytosis and the IL-18/IFN-γ–mediated molecular connection between innate and adaptive immunity in the lung
  Blood, 2002
• Granulocyte/Macrophage-Colony-stimulating Factor (GM-CSF) Regulates Lung Innate Immunity to Lipopolysaccharide through Akt/Erk Activation of NFκB and AP-1 in Vivo
  Journal of Biological Chemistry, 2002
• Human granulocyte-macrophage colony-stimulating factor induces expression of the tumor necrosis factor gene by the U937 cell line and by normal human monocytes.
  Journal of Clinical Investigation, 1987
• Purified colony-stimulating factors enhance the survival of human neutrophils and eosinophils in vitro: a rapid and sensitive microassay for colony-stimulating factors
  Blood, 1986
• The nature and action of granulocyte-macrophage colony stimulating factors
  Blood, 1980