GAP-43 and p75NGFR immunoreactivity in presynaptic cells following neuromuscular blockade by botulinum toxin in rat
- 1 June 1994
- journal article
- Published by Springer Nature in Journal of Neurocytology
- Vol. 23 (6) , 354-363
- https://doi.org/10.1007/bf01666525
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Calcitonin gene-related peptide-like immunoreactivity, in botulinum toxin-paralysed rat musclesNeuromuscular Disorders, 1994
- Elimination of superfluous neuromuscular junctions in rat calf muscles recovering from botulinum toxin–induced paralysisMuscle & Nerve, 1994
- Electron microscopic immunocytochemistry of GAP-43 within proximal and chronically denervated distal stumps of transected peripheral nerveJournal of Neurocytology, 1992
- Induction of nerve growth factor receptor (p75NGFr) mRNA within hypoglossal motoneurons following axonal injuryMolecular Brain Research, 1992
- The downregulation of growth-associated proteins in motoneurons at the onset of synapse elimination is controlled by muscle activity and IGF1Journal of Neuroscience, 1992
- Nerve sprouting in innervated adult skeletal muscle induced by exposure to elevated levels of insulin-like growth factors.The Journal of cell biology, 1990
- mRNA levels of all three neurofilament proteins decline following nerve transectionMolecular Brain Research, 1988
- Differential regulation of mRNA encoding nerve growth factor and its receptor in rat sciatic nerve during development, degeneration, and regeneration: role of macrophages.Proceedings of the National Academy of Sciences, 1987
- Changes of nerve growth factor synthesis in nonneuronal cells in response to sciatic nerve transection.The Journal of cell biology, 1987
- Changes in motor innervation and cholinesterase localization induced by botulinum toxin in skeletal muscle of the mouse: differences between fast and slow muscles.Journal of Neurology, Neurosurgery & Psychiatry, 1970