Activated neutrophils aggravate endothelial dysfunction after reperfusion of the ischemic feline myocardium
- 1 June 1992
- journal article
- Published by Elsevier in American Heart Journal
- Vol. 123 (6) , 1464-1471
- https://doi.org/10.1016/0002-8703(92)90796-x
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Neutrophil-mediated vasoconstriction and endothelial dysfunction in low-flow perfusion-reperfused cat coronary artery.Circulation Research, 1991
- Role of endothelial dysfunction in the pathogenesis of reperfusion injury after myocardial ischemiaThe FASEB Journal, 1991
- Time course of endothelial dysfunction and myocardial injury during myocardial ischemia and reperfusion in the cat.Circulation, 1990
- Effect of ischemia reperfusion or hypoxia reoxygenation on lung vascular permeability and resistanceJournal of Applied Physiology, 1990
- The role of the neutrophil and free radicals in ischemic myocardial injuryJournal of Molecular and Cellular Cardiology, 1989
- Superoxide radical: A likely link between reperfusion injury and inflammationAdvances in Free Radical Biology & Medicine, 1986
- Demonstration of Depressed Polymorphonuclear Leukocyte Function in Nonviremic FeLV-Infected CatsCancer Investigation, 1985
- Nafazatrom-induced salvage of ischemic myocardium in anesthetized dogs is mediated through inhibition of neutrophil function.Circulation Research, 1985
- Differential sensitivity of canine cardiac sarcolemmal and microsomal enzymes to inhibition by free radical-induced lipid peroxidation.Circulation Research, 1984
- Free radicals in cerebral ischemia.Stroke, 1978