Inactivation of IL11 Signaling Causes Craniosynostosis, Delayed Tooth Eruption, and Supernumerary Teeth
Open Access
- 1 July 2011
- journal article
- Published by Elsevier in American Journal of Human Genetics
- Vol. 89 (1) , 67-81
- https://doi.org/10.1016/j.ajhg.2011.05.024
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- Human Genome Sequencing Using Unchained Base Reads on Self-Assembling DNA NanoarraysScience, 2010
- gp130 signaling in bone cell biology: Multiple roles revealed by analysis of genetically altered miceMolecular and Cellular Endocrinology, 2009
- Apc inhibition of Wnt signaling regulates supernumerary tooth formation during embryogenesis and throughout adulthoodDevelopment, 2009
- The molecular mechanism behind bone remodelling: a reviewClinical Oral Investigations, 2009
- STAT3 and STAT1 mediate IL-11–dependent and inflammation-associated gastric tumorigenesis in gp130 receptor mutant miceJournal of Clinical Investigation, 2008
- Unravelling the molecular control of calvarial suture fusion in children with craniosynostosisBMC Genomics, 2007
- Continuous tooth generation in mouse is induced by activated epithelial Wnt/β-catenin signalingProceedings of the National Academy of Sciences, 2006
- Osteoclast signalling pathwaysBiochemical and Biophysical Research Communications, 2004
- Identification of the domain in the human interleukin-11 receptor that mediates ligand binding11Edited by J. A. WellsJournal of Molecular Biology, 2001
- Site-directed mutagenesis by overlap extension using the polymerase chain reactionGene, 1989