Bile Acids Reduce the Apoptosis-Inducing Effects of Sodium Butyrate on Human Colon Adenoma (AA/C1) Cells: Implications for Colon Carcinogenesis

This publication has 22 references indexed in Scilit:

• Administration of an unconjugated bile acid increases duodenal tumors in a murine model of familial adenomatous polyposis
  Carcinogenesis: Integrative Cancer Research, 1999
• Sodium butyrate induces apoptosis in human colonic tumour cell lines in a p53‐independent pathway: Implications for the possible role of dietary fibre in the prevention of large‐bowel cancer
  International Journal of Cancer, 1993
• Diet and Risk of Colorectal Adenomas: Macronutrients, Cholesterol, and Fiber
  JNCI Journal of the National Cancer Institute, 1993
• Dietary Intake of Fiber and Decreased Risk of Cancers of the Colon and Rectum: Evidence From the Combined Analysis of 13 Case-Control Studies
  JNCI Journal of the National Cancer Institute, 1992
• Bile acids, non‐phorbol‐ester‐type tumor promoters, stimulate the phosphorylation of protein kinase C substrates in human platelets and colon cell line HT29
  International Journal of Cancer, 1992
• Faecal unconjugated bile acids in patients with colorectal cancer or polyps.
  Gut, 1992
• A genetic model for colorectal tumorigenesis
  Cell, 1990
• Bile acids, but not neutral sterols, are tumor promoters in the colon in man and in rodents.
  Environmental Health Perspectives, 1983
• Effect of pH on bile salt degradation by mixed fecal cultures
  Steroids, 1978
• Metabolic epidemiology of colon cancer:Fecal bile acids and neutral sterols in colon cancer patients and patients with adenomatous polyps
  Cancer, 1977