Transcriptional Repressors Are Increased in Pancreatic Islets of Type 2 Diabetic Rats
- 30 December 1998
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 253 (3) , 712-718
- https://doi.org/10.1006/bbrc.1998.9833
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Alterations in basal and glucose-stimulated voltage-dependent Ca2+ channel activities in pancreatic beta cells of non-insulin-dependent diabetes mellitus GK rats.Journal of Clinical Investigation, 1996
- Hyperresponse in calcium-induced insulin release from electrically permeabilized pancreatic islets of diabetic GK rats and its defective augmentation by glucoseDiabetologia, 1995
- Reduced sensitivity of dihydroxyacetone on ATP-sensitive K+ channels of pancreatic beta cells in GK ratsDiabetologia, 1994
- Impact of Diabetic Inheritance on Glucose Tolerance and Insulin Secretion in Spontaneously Diabetic GK-Wistar RatsDiabetes, 1994
- Glucose Sensitivity of ATP-Sensitive K+ Channels Is Impaired in β-Cells of the GK Rat: A New Genetic Model of NIDDMDiabetes, 1993
- Functional and metabolic perturbations in isolated pancreatic islets from the GK rat, a genetic model of noninsulin-dependent diabetes.Endocrinology, 1993
- β-Cell Insensitivity to Glucose in the GK Rat, a Spontaneous Nonobese Model for Type II DiabetesDiabetes, 1991
- Preferential Reduction of Insulin Production in Mouse Pancreatic Islets Maintained in Culture after Streptozotocin Exposure*Endocrinology, 1988
- An in vivo analysis of pancreatic protein and insulin biosynthesis in a rat model for non-insulin-dependent diabetes.Journal of Clinical Investigation, 1984
- Spontaneous Diabetes Produced by Selective Breeding of Normal Wistar RatsProceedings of the Japan Academy, 1975