Decreased Glutamic Acid Decarboxylase67 Messenger RNA Expression in a Subset of Prefrontal Cortical γ-Aminobutyric Acid Neurons in Subjects With Schizophrenia
Top Cited Papers
- 1 March 2000
- journal article
- research article
- Published by American Medical Association (AMA) in Archives of General Psychiatry
- Vol. 57 (3) , 237-245
- https://doi.org/10.1001/archpsyc.57.3.237
Abstract
Research from JAMA Psychiatry — Decreased Glutamic Acid Decarboxylase67 Messenger RNA Expression in a Subset of Prefrontal Cortical γ-Aminobutyric Acid Neurons in Subjects With SchizophreniaKeywords
This publication has 36 references indexed in Scilit:
- A decrease of reelin expression as a putative vulnerability factor in schizophreniaProceedings of the National Academy of Sciences, 1998
- The sexually dimorphic nucleus of the hypothalamus contains GABA neurons in rat and manBrain Research, 1996
- Local circuit neurons of the prefrontal cortex in schizophrenia: selective increase in the density of calbindin-immunoreactive neuronsPsychiatry Research, 1995
- Neural Circuitry of the Prefrontal Cortex in SchizophreniaArchives of General Psychiatry, 1995
- Gene Expression for Glutamic Acid Decarboxylase Is Reduced Without Loss of Neurons in Prefrontal Cortex of SchizophrenicsArchives of General Psychiatry, 1995
- Regionally selective deficits in uptake sites for glutamate and gamma-aminobutyric acid in the basal ganglia in schizophreniaPsychiatry Research, 1992
- Two human glutamate decarboxylases, 65-kDa GAD and 67-kDa GAD, are each encoded by a single gene.Proceedings of the National Academy of Sciences, 1992
- Increased GABAA receptor binding in superficial layers of cingulate cortex in schizophrenicsJournal of Neuroscience, 1992
- Evidence of glutamatergic deficiency in schizophreniaNeuroscience Letters, 1991
- Reduced GABA uptake sites in the temporal lobe in schizophreniaNeuroscience Letters, 1989