Regional Distribution of Amyloid-Bri Deposition and Its Association with Neurofibrillary Degeneration in Familial British Dementia

This publication has 47 references indexed in Scilit:

• Variant Alzheimer's disease with spastic paraparesis and cotton wool plaques is caused by PS-1 mutations that lead to exceptionally high amyloid-? concentrations
  Annals of Neurology, 2000
• Familial British dementia with amyloid angiopathy
  Brain, 2000
• Sequential phosphorylation of Tau by glycogen synthase kinase‐3β and protein kinase A at Thr212 and Ser214 generates the Alzheimer‐specific epitope of antibody AT100 and requires a paired‐helical‐filament‐like conformation
  European Journal of Biochemistry, 1998
• Alzheimer's soluble amyloid β is a normal component of human urine
  FEBS Letters, 1997
• Early phosphorylation of tau in Alzheimerʼs disease occurs at Ser-202 and is preferentially located within neurites
  NeuroReport, 1994
• An extensive network of PHFτ‐rich dystrophic neurites permeates neocortex and nearly all neuritic and diffuse amyloid plaques in Alzheimer disease
  FEBS Letters, 1994
• Neuropathological stageing of Alzheimer-related changes
  Acta Neuropathologica, 1991
• FAMILIAL CEREBRAL AMYLOID ANGIOPATHY WITH NONNEURITIC AMYLOID PLAQUE FORMATION
  Brain, 1990
• On areas of transition between entorhinal allocortex and temporal isocortex in the human brain. Normal morphology and lamina-specific pathology in Alzheimer's disease
  Acta Neuropathologica, 1985
• Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4
  Nature, 1970