The Function of HSP72 in Suppression of c-Jun N-terminal Kinase Activation Can Be Dissociated from Its Role in Prevention of Protein Damage
Open Access
- 1 July 1999
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 274 (29) , 20223-20228
- https://doi.org/10.1074/jbc.274.29.20223
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Proteasome Inhibitors Activate Stress Kinases and Induce Hsp72Journal of Biological Chemistry, 1998
- Mast cell tumor necrosis factor α production is regulated by MEK kinasesProceedings of the National Academy of Sciences, 1997
- c-Jun NH2-terminal Kinase-mediated Activation of Interleukin-1β Converting Enzyme/CED-3-like Protease during Anticancer Drug-induced ApoptosisJournal of Biological Chemistry, 1997
- The Role of c-Jun N-terminal Kinase (JNK) in Apoptosis Induced by Ultraviolet C and γ RadiationJournal of Biological Chemistry, 1996
- Sounding the Alarm: Protein Kinase Cascades Activated by Stress and InflammationJournal of Biological Chemistry, 1996
- Protein kinase cascades activated by stress and inflammatory cytokinesBioEssays, 1996
- The stress-activated protein kinase pathway mediates cell death following injury induced by cis-platinum, UV irradiation or heatCurrent Biology, 1996
- Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosisNature, 1996
- The regulation of AP-1 activity by mitogen-activated protein kinasesPhilosophical Transactions Of The Royal Society B-Biological Sciences, 1996
- Opposing Effects of ERK and JNK-p38 MAP Kinases on ApoptosisScience, 1995