Progressive Cardiac Dysfunction and Fibrosis in the Cardiomyopathic Hamster and Effects of Growth Hormone and Angiotensin-Converting Enzyme Inhibition
- 19 October 1999
- journal article
- other
- Published by Wolters Kluwer Health in Circulation
- Vol. 100 (16) , 1734-1743
- https://doi.org/10.1161/01.cir.100.16.1734
Abstract
Background—Growth hormone (GH) improves cardiac function in the rat with myocardial infarction, but its effects in a model of primary dilated cardiomyopathy have not been reported. GH effects were ...Keywords
This publication has 21 references indexed in Scilit:
- Angiotensin II blockade followed by growth hormone as adjunctive therapy after experimental myocardial infarctionJournal of Cardiac Failure, 1998
- Randomised, double-blind, placebo-controlled trial of human recombinant growth hormone in patients with chronic heart failure due to dilated cardiomyopathyThe Lancet, 1998
- Both hypertrophic and dilated cardiomyopathies are caused by mutation of the same gene, δ-sarcoglycan, in hamster: An animal model of disrupted dystrophin-associated glycoprotein complexProceedings of the National Academy of Sciences, 1997
- Identification of the Syrian hamster cardiomyopathy geneHuman Molecular Genetics, 1997
- A Preliminary Study of Growth Hormone in the Treatment of Dilated CardiomyopathyNew England Journal of Medicine, 1996
- Growth Hormone and the HeartEndocrine Reviews, 1994
- Cardiomyopathic hamsters, CHF 146 and CHF 147: a preliminary studyCanadian Journal of Physiology and Pharmacology, 1984
- Afterload mismatch and preload reserve: A conceptual framework for the analysis of ventricular functionProgress in Cardiovascular Diseases, 1976
- Wall stress and patterns of hypertrophy in the human left ventricle.Journal of Clinical Investigation, 1975
- Hereditary cardiomyopathy: A new disease modelAmerican Heart Journal, 1969