Histopathological and hemodynamic studies supporting hypoxia and vascular disruption as explanation to phenytoin teratogenicity
- 1 November 1992
- journal article
- research article
- Published by Wiley in Teratology
- Vol. 46 (5) , 485-497
- https://doi.org/10.1002/tera.1420460513
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Identical phalangeal defects induced by phenytoin and nifedipine suggest fetal hypoxia and vascular disruption behind phenytoin teratogenicityTeratology, 1992
- Relationship between uterine vascular clamping, vascular disruption syndrome, and cocaine teratogenicityTeratology, 1990
- Histological and in vitro studies supporting decreased uteroplacental blood flow as explanation for digital defects after administration of vasodilatorsTeratology, 1990
- Digital defects induced by vasodilating agents: Relationship to reduction in uteroplacental blood flowTeratology, 1989
- Effects of calcium entry blocker (nicardipine) tocolysis rhesus macaques: Fetal plasma concentrations and cardiorespiratory changesAmerican Journal of Obstetrics and Gynecology, 1987
- Nifedipine: Effects on fetal and maternal hemodynamics in pregnant sheepAmerican Journal of Obstetrics and Gynecology, 1987
- Hemodynamic effects of intravenous cocaine on the pregnant ewe and fetusAmerican Journal of Obstetrics and Gynecology, 1986
- Effects of Diltiazem Hydrochloride on Embryonic Development: Species Differences in the Susceptibility and Stage Specificity in Mice, Rats, and RabbitsOkajimas Folia Anatomica Japonica, 1975
- Influence of Currents on Form of SpongesScience, 1960
- Baroreceptor reflexes in new‐born rabbitsThe Journal of Physiology, 1960