Association of idiopathic venous thromboembolism with single point-mutation at Arg506 of factor V
- 1 June 1994
- journal article
- research article
- Published by Elsevier in The Lancet
- Vol. 343 (8912) , 1535-1536
- https://doi.org/10.1016/s0140-6736(94)92939-4
Abstract
No abstract availableKeywords
This publication has 10 references indexed in Scilit:
- Hypercoagulability -- A New Cofactor in the Protein C Anticoagulant PathwayNew England Journal of Medicine, 1994
- Resistance to Activated Protein C as a Basis for Venous ThrombosisNew England Journal of Medicine, 1994
- Inherited resistance to activated protein C iscorrected by anticoagulant cofactor activity found to be a property of factorV.Proceedings of the National Academy of Sciences, 1994
- Role of the membrane in the inactivation of factor Va by activated protein C.Journal of Biological Chemistry, 1993
- Venous thrombosis due to poor anticoagulant response to activated protein C: Leiden Thrombophilia StudyThe Lancet, 1993
- Anticoagulant protein C pathway defective in majority of thrombophilic patients [see comments]Blood, 1993
- Familial thrombophilia due to a previously unrecognized mechanism characterized by poor anticoagulant response to activated protein C: prediction of a cofactor to activated protein C.Proceedings of the National Academy of Sciences, 1993
- Inherited predisposition to thrombosisCell, 1993
- Storage conditions of blood samples and primer selection affect the yield of cDNA polymerase chain reaction products of hepatitis C virusJournal of Clinical Microbiology, 1992
- Deficiencies of Coagulation-Inhibiting and Fibrinolytic Proteins in Outpatients with Deep-Vein ThrombosisNew England Journal of Medicine, 1990