Raised [3H]-5-HT release and 45Ca2+ uptake in diazepam withdrawal: Inhibition by baclofen

This publication has 14 references indexed in Scilit:

• GABAB receptors are up-regulated by chronic treatment with lithium or carbamazepine. GABA hypothesis of affective disorders?
  Published by Elsevier ,2002
• Concurrent treatment with verapamil prevents diazepam withdrawapl-induced anxiety, in the absence of altered calcium flux in cortical synaptosomes
  Neuropharmacology, 1992
• Chronic exposure to anxiolytic drugs, working by different mechanisms causes up-regulation of dihydropyridine binding sites on cultured bovine adrenal chromaffin cells
  Neuropharmacology, 1991
• Nitrendipine decreases benzodiazepine withdrawal seizures but not the development of benzodiazepine tolerance or withdrawal signs
  British Journal of Pharmacology, 1990
• The history of benzodiazepine dependence: A review of animal studies
  Neuroscience & Biobehavioral Reviews, 1990
• Chronic diazepam treatment in rats causes long-lasting changes in central [3H]-5-hydroxytryptamine and [14C]-γ-aminobutyric acid release
  British Journal of Pharmacology, 1990
• GABAB receptors and their significance in mammalian pharmacology
  Trends in Pharmacological Sciences, 1989
• Reversal of increased anxiety during benzodiazepine withdrawal: Evidence for an anxiogenic endogenous ligand for the benzodiazepine receptor
  Brain Research Bulletin, 1988
• GABAA and GABAB receptor site distribution in the rat central nervous system
  Neuroscience, 1987
• Chronic antidepressants and GABA “B” receptors: A GABA hypothesis of antidepressant drug action
  Life Sciences, 1984