Allelic Deletion at 9p21-22 in Primary Cutaneous CD30+ Large Cell Lymphoma
- 1 December 2000
- journal article
- Published by Elsevier in Journal of Investigative Dermatology
- Vol. 115 (6) , 1104-1107
- https://doi.org/10.1046/j.1523-1747.2000.00197.x
Abstract
No abstract availableThis publication has 11 references indexed in Scilit:
- Ki-1 (CD30) Positive Cutaneous Anaplastic Large Cell LymphomasPublished by S. Karger AG ,2015
- Comparative analysis of histologicai and immunohistological features in mycosis fungoides and Sézary syndromeJournal of Cutaneous Pathology, 1998
- Loss of Tumor Suppressor Gene Expression in High-Grade But Not Low-Grade Non-Hodgkin’s LymphomasAmerican Journal of Clinical Pathology, 1998
- Overexpression of p53 Protein in Cutaneous T Cell Lymphoma: Relationship to Large Cell Transformation and Disease ProgressionJournal of Investigative Dermatology, 1998
- p53 protein expression in non‐Hodgkin's lymphomas is infrequently related to p53 gene mutationsPathology International, 1998
- p53 and bcl‐2 expression do not correlate with prognosis in primary cutaneous large T‐cell lymphomasJournal of Cutaneous Pathology, 1997
- Role of the INK4a Locus in Tumor Suppression and Cell MortalityCell, 1996
- Frequency of homozygous deletion at p16/CDKN2 in primary human tumoursNature Genetics, 1995
- Deletions of the cyclin-dependent kinase inhibitor genes p16INK4A and p15INK4B in non-Hodgkin's lymphomasBlood, 1995
- Construction of a 2.8-megabase yeast artificial chromosome contig and cloning of the human methylthioadenosine phosphorylase gene from the tumor suppressor region on 9p21.Proceedings of the National Academy of Sciences, 1995