Inhibition of K+ efflux prevents mitochondrial dysfunction, and suppresses caspase-3-, apoptosis-inducing factor-, and endonuclease G-mediated constitutive apoptosis in human neutrophils
- 31 December 2006
- journal article
- Published by Elsevier in Cellular Signalling
- Vol. 18 (12) , 2302-2313
- https://doi.org/10.1016/j.cellsig.2006.05.013
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- CpG motifs in bacterial DNA delay apoptosis of neutrophil granulocytesThe FASEB Journal, 2004
- Cytoprotective Role of Ca 2+ - Activated K + Channels in the Cardiac Inner Mitochondrial MembraneScience, 2002
- Loss of Pentameric Symmetry of C-reactive Protein Is Associated with Delayed Apoptosis of Human NeutrophilsJournal of Biological Chemistry, 2002
- p38 Mitogen‐activated protein kinase and phosphatidylinositol 3‐kinase activities have opposite effects on human neutrophil apoptosisThe FASEB Journal, 2001
- Physiological Concentrations of K+ Inhibit Cytochrome c-dependent Formation of the ApoptosomeJournal of Biological Chemistry, 2001
- Peroxynitrite induces integrin‐dependent adhesion of human neutrophils to endothelial cells via activation of the Raf‐1/MEK/Erk pathwayThe FASEB Journal, 2000
- Ca2+-Activated K Channel of the BK-Type in the Inner Mitochondrial Membrane of a Human Glioma Cell LineBiochemical and Biophysical Research Communications, 1999
- Role of Potassium Channels in Amyloid‐Induced Cell DeathJournal of Neurochemistry, 1998
- Cell shrinkage and apoptosis: a role for potassium and sodium ion effluxCell Death & Differentiation, 1997
- Voltage-dependent and Ca2(+)-activated ion channels in human neutrophils.Journal of Clinical Investigation, 1990