Cytochrome c Release Occurs via Ca2+-dependent and Ca2+-independent Mechanisms That Are Regulated by Bax

This publication has 39 references indexed in Scilit:

• Preservation of Mitochondrial Structure and Function after Bid- or Bax-Mediated Cytochrome c Release
  The Journal of cell biology, 2000
• Molecular Mechanisms of Apoptosis Induced by Cytotoxic Chemicals
  Critical Reviews in Toxicology, 2000
• The Release of Cytochrome c from Mitochondria during Apoptosis of NGF-deprived Sympathetic Neurons Is a Reversible Event
  The Journal of cell biology, 1999
• Bax and Adenine Nucleotide Translocator Cooperate in the Mitochondrial Control of Apoptosis
  Science, 1998
• Mitochondria and Apoptosis
  Science, 1998
• PK11195, a Ligand of the Mitochondrial Benzodiazepine Receptor, Facilitates the Induction of Apoptosis and Reverses Bcl-2-Mediated Cytoprotection
  Experimental Cell Research, 1998
• Multiple Conformations of Physiological Membrane-Bound Cytochrome c
  Biochemistry, 1998
• Release of Cytochrome c from Liver Mitochondria during Permeability Transition
  Biochemical and Biophysical Research Communications, 1997
• Prevention of Apoptosis by Bcl-2: Release of Cytochrome c from Mitochondria Blocked
  Science, 1997
• Hypotonic fragility of outer membrane and activation of external pathway of NADH oxidation in rat liver mitochondria are increased with age
  Mechanisms of Ageing and Development, 1993