Chorionic Gonadotropin in Obesity

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Abstract
After 40 years of trying every new approach to the treatment of obesity with little or no success, I believe a new method that works has been made available to us. It works with about 60–70% of obese patients of both sexes aged 15 and up, provided the method is followed meticulously as the author has developed it. No one can yet say for certain how or why it works, and a great deal of research will be needed. I hope that this report of my own clinical findings together with some possible tentative explanations will act as a stimulus for this research. While it is true that obesity is due to excessive calorie intake, this tells us nothing of the basic causes. What needs further explanation and discovery are the mechanisms that regulate calorie intake and output as well as the reason for the failure of these regulating mechanisms in some individuals that results in obesity. Recently, Dr. Margaret Albrink (34) has made a very interesting observation that while man has elaborate and efficient mechanisms for surviving starvation, his techniques for handling a surplus of food—a frequent necessity in modern societies of abundance—are limited and easily saturated. As a result, she suggests, the metabolic abnormalities leading to atherosclerosis may be a result of over-burdening the mechanisms for storage of fuel, i.e., overstuffing the adipose cell. It is intriguing to speculate on a parallel explanation for the metabolic abnormality that leads to obesity. Further weight to this possible explanation is brought to bear by the fact that the adipose cell in the obese has been found to be larger than the normal fat cell. It follows, therefore, that at least two types of fat cells exist, the normal cell storing the reserve fuel that is necessary for good health and the abnormally large cell that becomes overstuffed and forms the excess fat tissue of the obese. This approach to obesity also lends further credence to the belief that the manner in which food is utilized and stored in the body is a more complicated mechanism than previously believed and that there is a controlling center in the brain. Medicine must not neglect nor abandon the problem of obesity for many reasons, not the least of which is its relationship to cancer and the process of aging. A. Tanenbaum and H. Silverstone (35) suggest that nutrition plays a role in the formation of cancer in man. “When living cells are subjected to carcinogenic influences they may undergo changes that finally result in growing neoplasm. The energy and substance for the development of the first cancer cells are derived principally from the animal; the new cell type increases in number by assimilating nutrients from the host. It may be expected then that the diet and the nutritional state of the host influence the formation and the growth of tumors.” The process of aging as defined by Howard J. Curtis of the Brookhaven National Laboratory “may be considered as an increasing probability of developing a degenerative disease.” Furthermore, M. H. Ross (36) in his experimental studies with rodents has shown that a calorie-restricted diet delays the onset of development of all the degenerative diseases in rodents (and presumably in man) and obesity speeds their development. Medicine must neither accept nor abandon Dr. Simeons’ method until it has been properly tested through research or until a completely different and a better method is discovered. Dr. Simeons himself has begun such testing and research in his clinic. He has often told me in person and has more than once said publicly that critics and researchers in this field are welcome at his Clinic to study his results and observations. Let us hope some of them will accept his offer soon.

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