LIS1 and dynein motor function in neuronal migration and development
Open Access
- 15 March 2001
- journal article
- review article
- Published by Cold Spring Harbor Laboratory in Genes & Development
- Vol. 15 (6) , 639-651
- https://doi.org/10.1101/gad.886801
Abstract
A biweekly scientific journal publishing high-quality research in molecular biology and genetics, cancer biology, biochemistry, and related fieldsThis publication has 86 references indexed in Scilit:
- Microtubules and Neuronal PolarityNeuron, 1999
- Reeler: new tales on an old mutant mouseBioEssays, 1998
- Point Mutations and an Intragenic Deletion in LIS1, the Lissencephaly Causative Gene in Isolated Lissencephaly Sequence and Miller-Dieker SyndromeHuman Molecular Genetics, 1997
- A Revision of the Lissencephaly and Miller-Dieker Syndrome Critical Regions in Chromosome 17p13.3Human Molecular Genetics, 1997
- The reeler gene encodes a protein with an EGF–like motif expressed by pioneer neuronsNature Genetics, 1995
- Binding of Pleckstrin Homology Domains to WD40/β-Transducin Repeat Containing Segments of the Protein Product of the Lis-1 GeneBiochemical and Biophysical Research Communications, 1995
- A protein related to extracellular matrix proteins deleted in the mouse mutant reelerNature, 1995
- New insights into the interaction of cytoplasmic dynein with the actin-related protein, Arp1.The Journal of cell biology, 1994
- The ancient regulatory-protein family of WD-repeat proteinsNature, 1994
- A vertebrate actin-related protein is a component of a multisubunit complex involved in microtubule-based vesicle motilityNature, 1992