Locomotor hyperactivity and alterations in dopamine neurotransmission are associated with overexpression of A53T mutant human α-synuclein in mice
- 14 October 2005
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 21 (2) , 431-443
- https://doi.org/10.1016/j.nbd.2005.08.005
Abstract
No abstract availableKeywords
This publication has 47 references indexed in Scilit:
- Chronoamperometry To Determine Differential Reductions in Uptake in Brain Synaptosomes from Serotonin Transporter Knockout MiceAnalytical Chemistry, 2004
- Tauists, Baptists, Syners, Apostates, and new dataAnnals of Neurology, 2002
- Mice Lacking α-Synuclein Display Functional Deficits in the Nigrostriatal Dopamine SystemNeuron, 2000
- Binding of α-Synuclein to Brain Vesicles Is Abolished by Familial Parkinson's Disease MutationJournal of Biological Chemistry, 1998
- Stabilization of α-Synuclein Secondary Structure upon Binding to Synthetic MembranesJournal of Biological Chemistry, 1998
- AlaSOPro mutation in the gene encoding α-synuclein in Parkinson's diseaseNature Genetics, 1998
- Mutation in the α-Synuclein Gene Identified in Families with Parkinson's DiseaseScience, 1997
- Drug addiction: Knockout mice and dirty drugsCurrent Biology, 1996
- Identification of two distinct synucleins from human brainFEBS Letters, 1994
- Autoradiographic localization of D1 dopamine receptors in the rat brain with [3H]SCH 23390Brain Research, 1986