Cell Cycle Arrest and Cell Death Are Controlled by p53-dependent and p53-independent Mechanisms in Tsg101-deficient Cells
Open Access
- 1 August 2004
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 279 (34) , 35984-35994
- https://doi.org/10.1074/jbc.m400408200
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Tsg101 Is Essential for Cell Growth, Proliferation, and Cell Survival of Embryonic and Adult TissuesMolecular and Cellular Biology, 2003
- Targeted Deletion of the Tsg101 Gene Results in Cell Cycle Arrest at G1/S and p53-independent Cell DeathJournal of Biological Chemistry, 2002
- Genomic architecture and transcriptional activation of the mouse and human tumor susceptibility gene TSG101: Common types of shorter transcripts are true alternative splice variantsOncogene, 1998
- A Putative Tumor Suppressor, TSG101, Acts as a Transcriptional Suppressor through Its Coiled-Coil DomainBiochemical and Biophysical Research Communications, 1998
- Interlayer Porosity in Montmorillonite Intercalated with Keggin-like Cation Studied by Molecular Mechanics SimulationJournal of Molecular Modeling, 1997
- TSG101 may be the prototype of a class of dominant negative ubiquitin regulatorsNature Genetics, 1997
- RETRACTED: The TSG101 Tumor Susceptibility Gene Is Located in Chromosome 11 Band p15 and Is Mutated in Human Breast CancerCell, 1997
- p21 is a universal inhibitor of cyclin kinasesNature, 1993
- WAF1, a potential mediator of p53 tumor suppressionCell, 1993
- Evaluation of glucocorticoid‐induced DNA fragmentation in mouse thymocytes by flow cytometryCell Proliferation, 1991