Nucleotide Excision Repair, Oxidative Damage, DNA Sequence Polymorphisms, and Cancer Treatment
Open Access
- 15 February 2005
- journal article
- editorial
- Published by American Association for Cancer Research (AACR) in Clinical Cancer Research
- Vol. 11 (4) , 1355-1357
- https://doi.org/10.1158/1078-0432.ccr-05-0024
Abstract
Can a single polymorphic site help tailor a cancer patient's chemotherapy regimen? The study by Zhao et al. [(1)][1] presented in this issue of Clinical Cancer Research suggests that in fact polymorphic sites in noncoding gene regions may determine how a patient will react to a given chemotherapyKeywords
This publication has 16 references indexed in Scilit:
- Nucleotide Excision Repair Gene Polymorphisms and Recurrence after Treatment for Superficial Bladder CancerClinical Cancer Research, 2005
- Nucleotide Excision Repair in E. Coli and ManAdvances in Protein Chemistry, 2004
- Molecular Mechanisms of Mammalian DNA Repair and the DNA Damage CheckpointsAnnual Review of Biochemistry, 2004
- Base Excision RepairAdvances in Protein Chemistry, 2004
- Recognition and repair of the cyclobutane thymine dimer, a major cause of skin cancers, by the human excision nucleaseGenes & Development, 2003
- DNA Repair Excision Nuclease Attacks Undamaged DNAJournal of Biological Chemistry, 2001
- Identification of four single nucleotide polymorphisms in DNA repair genes:XPA andXPB(ERCC3) in Polish populationHuman Mutation, 2000
- Nucleotide Excision Repair in Mammalian CellsJournal of Biological Chemistry, 1997
- DNA EXCISION REPAIRAnnual Review of Biochemistry, 1996
- Substrate spectrum of human excinuclease: repair of abasic sites, methylated bases, mismatches, and bulky adducts.Proceedings of the National Academy of Sciences, 1994