THE HUMAN PYRAMIDAL TRACT
- 1 November 1945
- journal article
- other
- Published by American Medical Association (AMA) in Archives of Neurology & Psychiatry
- Vol. 54 (5) , 339-343
- https://doi.org/10.1001/archneurpsyc.1945.02300110023003
Abstract
The purpose of the present investigation is to correlate as nearly as possible the motor deficits produced in striated muscles by cerebrovascular lesions with axonal destruction in the pyramids. Such injuries, in the form of emboli, thromboses or hemorrhages, are known to have a destructive action on nerve tissue. It may be possible that these three forms of vascular disturbance do not act in exactly the same manner on the neurons of the pyramidal tract. Wide variations in the extent and potency of each type may be the rule. Although it appears that the true status of the cerebral collateral circulation has not been definitely settled, this vascular supply may be a factor in maintaining the viability of neurons under certain pathologic conditions, especially those of the thrombotic type. All of these factors may be reflected in the damage done to the axons of the pyramidal tract. The pyramidal fasciculusKeywords
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