Tumor Necrosis Factor-α and CD80 Modulate CD28 Expression through a Similar Mechanism of T-cell Receptor-independent Inhibition of Transcription
Open Access
- 1 July 2004
- journal article
- Published by Elsevier
- Vol. 279 (28) , 29130-29138
- https://doi.org/10.1074/jbc.m402194200
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- Molecular Basis for the Loss of CD28 Expression in Senescent T CellsPublished by Elsevier ,2002
- The B7–CD28 superfamilyNature Reviews Immunology, 2002
- Cellular senescence as a tumor-suppressor mechanismPublished by Elsevier ,2001
- Cellular senescence as a tumor-suppressor mechanismTrends in Cell Biology, 2001
- Comparison of CD8+ T-cell subsets in HIV-infected rapid progressor children versus non–rapid progressor childrenJournal of Allergy and Clinical Immunology, 2001
- Telomere Shortening and Decreased Replicative Potential, Contrasted by Continued Proliferation of Telomerase-Positive CD8+CD28lo T Cells in Patients with Systemic Lupus ErythematosusClinical Immunology, 2001
- CD80 and CD86 Are Not Equivalent in Their Ability to Induce the Tyrosine Phosphorylation of CD28Journal of Biological Chemistry, 1999
- CD28/B7 SYSTEM OF T CELL COSTIMULATIONAnnual Review of Immunology, 1996
- Analysis of T-lymphocyte subpopulations in inflammatory bowel diseases by three-color flow cytometryDigestive Diseases and Sciences, 1994
- CD28 interaction with B7 costimulates primary allogeneic proliferative responses and cytotoxicity mediated by small, resting T lymphocytes.The Journal of Experimental Medicine, 1992