Expression of c-Fos protein immunoreactivity in rat brain during ethanol withdrawal is prevented by nifedipine
- 1 January 1998
- Vol. 15 (1) , 71-76
- https://doi.org/10.1016/s0741-8329(97)00100-6
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- Excitotoxicity and Alcohol‐Related Brain DamageAlcohol, Clinical and Experimental Research, 1993
- NMDA and non-NMDA receptor-mediated increase of c-fos mRNA in dentate gyrus neurons involves calcium influx via different routesJournal of Neuroscience, 1992
- Regional induction of c-fos mRNA by NMDANeuroReport, 1991
- Effects of dihydropyridine calcium channel antagonists in ethanol withdrawal; doses required, stereospecificity and actions of Bay K 8644Psychopharmacology, 1990
- Ethanol withdrawal seizures and the NMDA receptor complexEuropean Journal of Pharmacology, 1990
- Localization and induction of c-fos protein-like immunoreactive material in the nuclei of adult mammalian neuronsBrain Research, 1988
- Increased dihydropyridine-sensitive calcium channels in rat brain may underlie ethanol physical dependenceNeuropharmacology, 1987
- Calcium channel antagonists decrease the ethanol withdrawal syndromeLife Sciences, 1986
- Stimulation of Neuronal Acetylcholine Receptors Induces Rapid Gene TranscriptionScience, 1986
- Altered characteristics of [3H]dopamine release from superfused slices of corpus striatum obtained from rats receiving ethanol in vivoNeuropharmacology, 1985