Overexpression of Bcl-2 protects human hepatoma cells from Fasantibody-mediated apoptosis
- 31 August 1999
- journal article
- Published by Elsevier in Journal of Hepatology
- Vol. 31 (2) , 315-322
- https://doi.org/10.1016/s0168-8278(99)80230-x
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- Involvement of MACH, a Novel MORT1/FADD-Interacting Protease, in Fas/APO-1- and TNF Receptor–Induced Cell DeathCell, 1996
- Involvement of the CD95 (APO-1/Fas) receptor and ligand in liver damage.The Journal of Experimental Medicine, 1995
- BCL-2 protects from lethal hepatic apoptosis induced by the anti-FAS antibody in mice U. ICGM, Universit Ren Descartes, Paris, FranceHepatology, 1995
- Immunohistochemical detection of Fas antigen in liver tissue of patients with chronic hepatitis CHepatology, 1994
- Bcl-2 and the regulation of programmed cell deathThe Journal of cell biology, 1994
- Lethal effect of the anti-Fas antibody in miceNature, 1993
- Discordant estimates of heterologous promoter activity as determined by reporter gene mRNA levels and enzyme activityBiochemical and Biophysical Research Communications, 1992
- The polypeptide encoded by the cDNA for human cell surface antigen Fas can mediate apoptosisCell, 1991
- Controlled synthesis of HBsAg in a differentiated human liver carcinoma-derived cell lineNature, 1979