Does the formation of lipopolysaccharide tolerance require intact vagal innervation of the liver?
- 20 December 2000
- journal article
- Published by Elsevier in Autonomic Neuroscience
- Vol. 85 (1) , 111-118
- https://doi.org/10.1016/s1566-0702(00)00229-0
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Nuclear Factor-κB — A Pivotal Transcription Factor in Chronic Inflammatory DiseasesNew England Journal of Medicine, 1997
- Subdiaphragmatic vagotomy inhibits intra-abdominal interleukin-1β stimulation of adrenocorticotropin secretionBrain Research, 1996
- Subdiaphragmatic vagotomy suppresses endotoxin-induced activation of hypothalamic corticotropin-releasing hormone neurons and ACTH secretionEndocrinology, 1995
- The nonspecific nature of endotoxin toleranceTrends in Microbiology, 1995
- Interleukin-1β induced corticosterone elevation and hypothalamic NE depletion is vagally mediatedBrain Research Bulletin, 1995
- Experimental basis for neural-immune interactionsPhysiological Reviews, 1995
- Glucocorticoid‐dependent and ‐independent mechanisms involved in lipopolysaccharide toleranceEuropean Journal of Immunology, 1991
- Metabolic Fate of Endotoxin in RatPublished by Springer Nature ,1990
- Energy balance and brown adipose tissue thermogenesis during chronic endotoxemia in ratsJournal of Applied Physiology, 1989
- TOLERANCE TO BACTERIAL PYROGENSThe Journal of Experimental Medicine, 1947