Seladin-1 transcription is linked to neuronal degeneration in Alzheimer’s disease
- 1 August 2002
- journal article
- Published by Elsevier in Neuroscience
- Vol. 113 (2) , 301-310
- https://doi.org/10.1016/s0306-4522(02)00180-x
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Inhibition of PP‐2A upregulates CaMKII in rat forebrain and induces hyperphosphorylation of tau at Ser 262/356FEBS Letters, 2001
- Phosphorylation of Microtubule-associated Protein Tau Is Regulated by Protein Phosphatase 2A in Mammalian BrainJournal of Biological Chemistry, 2000
- β-Amyloid Load Is Not Influenced by the Severity of Cardiovascular Disease in Aged and Demented PatientsStroke, 1999
- A novel oxidoreductase family sharing a conserved FAD-binding domainTrends in Biochemical Sciences, 1998
- Neuronal loss correlates with but exceeds neurofibrillary tangles in Alzheimer's diseaseAnnals of Neurology, 1997
- Familial Alzheimer's Disease–Linked Presenilin 1 Variants Elevate Aβ1–42/1–40 Ratio In Vitro and In VivoNeuron, 1996
- Profound Loss of Layer II Entorhinal Cortex Neurons Occurs in Very Mild Alzheimer’s DiseaseJournal of Neuroscience, 1996
- Neuropathological stageing of Alzheimer-related changesActa Neuropathologica, 1991
- Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's diseaseNature, 1991
- The Topographical and Neuroanatomical Distribution of Neurofibrillary Tangles and Neuritic Plaques in the Cerebral Cortex of Patients with Alzheimer's DiseaseCerebral Cortex, 1991