Inhibition of NADPH Oxidase is Neuroprotective after Ischemia—Reperfusion
Top Cited Papers
Open Access
- 6 May 2009
- journal article
- Published by SAGE Publications in Journal of Cerebral Blood Flow & Metabolism
- Vol. 29 (7) , 1262-1272
- https://doi.org/10.1038/jcbfm.2009.47
Abstract
Nicotinamide adenine dinucleotide phosphate (NADPH) oxidase (NOX) is well known as a major source for superoxide radical generation in leukocytes. Superoxide radicals play a significant role in bra...Keywords
This publication has 39 references indexed in Scilit:
- Gene inactivation of Na+/H+ exchanger isoform 1 attenuates apoptosis and mitochondrial damage following transient focal cerebral ischemiaEuropean Journal of Neuroscience, 2008
- Apocynin improves outcome in experimental stroke with a narrow dose rangeNeuroscience, 2008
- Apocynin, NADPH Oxidase, and Vascular CellsHypertension, 2008
- Apocynin Is Not an Inhibitor of Vascular NADPH Oxidases but an AntioxidantHypertension, 2008
- Hypoglycemic neuronal death is triggered by glucose reperfusion and activation of neuronal NADPH oxidaseJournal of Clinical Investigation, 2007
- Reduced Oxidative Stress Promotes NF-κB-Mediated Neuroprotective Gene Expression after Transient Focal Cerebral Ischemia: Lymphocytotrophic Cytokines and Antiapoptotic FactorsJournal of Cerebral Blood Flow & Metabolism, 2007
- The inflammatory response in strokeJournal of Neuroimmunology, 2006
- Exacerbation of Delayed Cell Injury After Transient Global Ischemia in Mutant Mice With CuZn Superoxide Dismutase DeficiencyStroke, 1999
- Role of Oxidants in Ischemic Brain DamageStroke, 1996
- Human copper-zinc superoxide dismutase transgenic mice are highly resistant to reperfusion injury after focal cerebral ischemia.Stroke, 1994