Inhibition of leukotriene b4 formation in human neutrophils after oral nafazatrom (Bay g 6575)
- 1 June 1985
- journal article
- research article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 34 (11) , 1891-1894
- https://doi.org/10.1016/0006-2952(85)90304-1
Abstract
No abstract availableThis publication has 17 references indexed in Scilit:
- The biologically active leukotrienes. Biosynthesis, metabolism, receptors, functions, and pharmacology.Journal of Clinical Investigation, 1984
- Prostaglandin I3 is formed in vivo in man after dietary eicosapentaenoic acidNature, 1984
- Studies on the mechanism of leukotriene induced coronary artery constrictionProstaglandins, 1983
- The influence of selective thromboxane synthetase inhibition with a novel imidazole derivative, UK-38,485, on prostanoid formation in man.Circulation, 1983
- THE PHARMACOLOGY AND PATHOPHYSIOLOGY OF LEUKOTRIENE B4British Medical Bulletin, 1983
- Reaction of the antithrombotic and antimetastatic agent, nafazatrom, with oxidizing radicalsBiochemical and Biophysical Research Communications, 1983
- In vivo and in vitro effects of nafazatrom (Bay g 6575), an antithrombotic compound, on arachidonic acid metabolism in platelets and vascular tissueBiochemical Pharmacology, 1983
- Characterization of leukotriene A4 and B4 bioysnthesisProstaglandins, 1983
- Nafazatrom (bay g 6575) inhibition of tumor cell lipoxygenase activity and cellular proliferationFEBS Letters, 1982
- A rapid method for purification of human granulocytes using percoll®. A comparison with dextran sedimentationJournal of Immunological Methods, 1981