Human mAChR antibodies from Sjögren syndrome sera increase cerebral nitric oxide synthase activity and nitric oxide synthase mRNA level
- 9 September 2004
- journal article
- Published by Elsevier in Clinical Immunology
- Vol. 113 (2) , 193-202
- https://doi.org/10.1016/j.clim.2004.08.005
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Role of Salivary IgA in the Pathogenesis of Sjögren SyndromeClinical Immunology, 2002
- Impaired microvascular response to cholinergic stimuli in primary Sjögren's syndromeAnnals of the Rheumatic Diseases, 2000
- Activation of Nitric Oxide Signaling through Muscarinic Receptors in Submandibular Glands by Primary Sjögren Syndrome AntibodiesClinical Immunology, 1999
- An Alternative Perspective to the Immune Response in Autoimmune Exocrinopathy: Induction of Functional Quiescence Rather Than Destructive AutoaggressionScandinavian Journal of Immunology, 1999
- Evolving concepts of diagnosis, pathogenesis, and therapy of Sj??gren??s syndromeCurrent Opinion in Rheumatology, 1998
- Desensitization and Sequestration of Human m2 Muscarinic Acetylcholine Receptors by Autoantibodies from Patients with Chagas' DiseaseJournal of Biological Chemistry, 1997
- Circulating antibodies against rat parotid gland M3 muscarinic receptors in primary Sjögren’s syndromeClinical and Experimental Immunology, 1996
- Alterations in the Secretory Response of Non-obese Diabetic (NOD) Mice to Muscarinic Receptor StimulationClinical Immunology and Immunopathology, 1996
- Turning off the signal: desensitization of β‐adrenergic receptor functionThe FASEB Journal, 1990
- SJÖGREN'S SYNDROME: ASSOCIATION WITH TYPE-1 DIABETES MELLITUSRheumatology, 1989