Protein Kinase C-α Activity Modulates Transepithelial Permeability and Cell Junctions in the LLC-PK1 Epithelial Cell Line
Open Access
- 1 June 1997
- journal article
- Published by Elsevier
- Vol. 272 (23) , 14950-14953
- https://doi.org/10.1074/jbc.272.23.14950
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Chronic Exposure of LLC-PK1Epithelia to the Phorbol Ester TPA Produces Polyp-like Foci with Leaky Tight Junctions and Altered Protein Kinase C-α Expression and LocalizationExperimental Cell Research, 1996
- Protein Kinase C-θ Isoenzyme Selective Stimulation of the Transcription Factor Complex AP-1 in T LymphocytesMolecular and Cellular Biology, 1996
- Protein Kinase C: Structure, Function, and RegulationJournal of Biological Chemistry, 1995
- The Protein Kinase C Inhibitor, Bisindolylmaleimide, Inhibits the TPA-Induced But Not the TNF-Induced Increase in LLC-PK1 Transepithelial PermeabilityBiochemical and Biophysical Research Communications, 1995
- Protein kinase C - a question of specificityTrends in Biochemical Sciences, 1994
- Assembly of the tight junction: the role of diacylglycerol.The Journal of cell biology, 1993
- Tight control of gene expression in mammalian cells by tetracycline-responsive promoters.Proceedings of the National Academy of Sciences, 1992
- The role of phosphorylation in development of tight junctions in cultured renal epithelial (MDCK) cellsBiochemical and Biophysical Research Communications, 1991
- A novel phorbol ester receptor/protein kinase, nPKC, distantly related to the protein kinase C familyCell, 1988
- Effects of tumor promoters on LLC-PK1 renal epithelial tight junctions and transepithelial fluxesAmerican Journal of Physiology-Cell Physiology, 1986