An ATPase/Helicase Complex Is an Essential Cofactor for Oncogenic Transformation by c-Myc

This publication has 44 references indexed in Scilit:

• The Myc oncoprotein: a critical evaluation of transactivation and target gene regulation
  Oncogene, 1999
• c-myc null cells misregulate cad and gadd45 but not other proposed c-Myc targets
  Genes & Development, 1998
• Pontin52, an interaction partner of β-catenin, binds to the TATA box binding protein
  Proceedings of the National Academy of Sciences, 1998
• The NOT, SPT3, and MOT1 Genes Functionally Interact To Regulate Transcription at Core Promoters
  Molecular and Cellular Biology, 1996
• RSC, an Essential, Abundant Chromatin-Remodeling Complex
  Cell, 1996
• An Essential Domain of the c-Myc Protein Interacts with a Nuclear Factor That Is Also Required for E1A-Mediated Transformation
  Molecular and Cellular Biology, 1995
• Mot1, a global repressor of RNA polymerase II transcription, inhibits TBP binding to DNA by an ATP-dependent mechanism.
  Genes & Development, 1994
• The ornithine decarboxylase gene is a transcriptional target of c-Myc.
  Proceedings of the National Academy of Sciences, 1993
• Oncogenic activity of the c-Myc protein requires dimerization with Max
  Cell, 1993
• THE myc ONCOGENE: ITS ROLE IN TRANSFORMATION AND DIFFERENTIATION
  Annual Review of Genetics, 1986