Dose-Dependent Acceleration of High-Density Lipoprotein Catabolism by Endothelial Lipase

Abstract
Background— Factors that regulate the metabolism of HDL and apolipoprotein A-I (apoA-I) are incompletely understood. Overexpression of endothelial lipase (EL) markedly reduces plasma levels of HDL cholesterol and apoA-I in mice, but the mechanisms of this effect remain unknown.

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