Cytochrome aa3 depletion is the cause of the deficient mitochondrial respiration induced by chronic valproate administration
- 1 February 1992
- journal article
- research article
- Published by Elsevier in Biochemical Pharmacology
- Vol. 43 (3) , 644-647
- https://doi.org/10.1016/0006-2952(92)90590-f
Abstract
No abstract availableKeywords
This publication has 16 references indexed in Scilit:
- In vitro effects of eight-carbon fatty acids on oxidations in rat liver mitochondriaBiochemical Pharmacology, 1990
- Valproic acid hepatic fatalitiesNeurology, 1987
- Influence of valproic acid on hepatic carbohydrate and lipid metabolismArchives of Biochemistry and Biophysics, 1983
- Valproate–Induced Hepatic Injury: Analyses of 23 Fatal CasesHepatology, 1982
- Inhibitory effects of sodium valproate on oxidative phosphorylationNeurology, 1981
- The occurrence of C6–C10-dicarboxylic acids in urine from patients and rats treated with dipropylacetateBiochemical Medicine, 1980
- Reye-like syndrome associated with valproic acid therapyThe Journal of Pediatrics, 1979
- A spectrophotometric procedure for rapid and sensitive measurements of β-oxidation. Demonstration of factors that can be rate-limiting for β-oxidationBiochemical Journal, 1977
- Sodium ValproateDrugs, 1977
- Molecular proportion of the fixed cytochrome components of the respiratory chain of Keilin-Hartree particles and beef heart mitochondriaBiochimica et Biophysica Acta (BBA) - Enzymology and Biological Oxidation, 1966