Wiedemann‐Beckwith syndrome, imprinting, IGF2, and H19: Implications for hemihyperplasia, associated neoplasms, and overgrowth
- 15 August 1994
- journal article
- Published by Wiley in American Journal of Medical Genetics
- Vol. 52 (2) , 233-234
- https://doi.org/10.1002/ajmg.1320520222
Abstract
No abstract availableThis publication has 16 references indexed in Scilit:
- Sex dependent transmission of Beckwith-Wiedemann syndrome associated with a reciprocal translocation t(9;11)(p11.2;p15.5).Journal of Medical Genetics, 1993
- Disruption of insulin–like growth factor 2 imprinting in Beckwith–Wiedemann syndromeNature Genetics, 1993
- Beckwith-wiedemann syndrome, tumourigenesis and imprintingCurrent Opinion in Genetics & Development, 1992
- Physical linkage of two mammalian imprinted genes, H19 and insulin–like growth factor 2Nature Genetics, 1992
- Evidence for paternal imprinting in familial Beckwith-Wiedemann syndrome.Journal of Medical Genetics, 1992
- Dads and disomy and diseaseNature, 1991
- Uniparental paternal disomy in a genetic cancer-predisposing syndromeNature, 1991
- Parental imprinting of the mouse H19 geneNature, 1991
- Tumours and hemihypertrophy associated with Wiedemann-Beckwith syndromeEuropean Journal of Pediatrics, 1983
- Complete and incomplete forms of Beckwith-Wiedemann syndrome: Their oncogenic potentialThe Journal of Pediatrics, 1980