On the Mechanism of Accumulation of 67Ga by Tumors

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Abstract
There is strong experimental evidence indicating that competitive binding of 67Ga3+ to Ca2+- and Mg2+ binding sites rather than a metabolic process is involved in the accumulation of this radioisotope. These observations suggest that in mouse BW5147 lymphatic leukemia and in other inflammatory and infectious processes, the most important role in 67Ga accumulation is played by a higher availability of those binding sites than in normal tissue.

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