Down-Regulation of p27 Is Associated with Development of Colorectal Adenocarcinoma Metastases
Open Access
- 30 September 1998
- journal article
- research article
- Published by Elsevier in The American Journal of Pathology
- Vol. 153 (3) , 681-687
- https://doi.org/10.1016/s0002-9440(10)65610-6
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (GM/CA 57587–01, CA 76584–01A1, CA 44704–09)
This publication has 45 references indexed in Scilit:
- Cytoplasmic displacement of cyclin E-cdk2 inhibitors p21Cip1 and p27Kip1 in anchorage-independent cellsOncogene, 1998
- Cancer Cell CyclesScience, 1996
- Enhanced Growth of Mice Lacking the Cyclin-Dependent Kinase Inhibitor Function of p27Cell, 1996
- Mice Lacking p27 Display Increased Body Size, Multiple Organ Hyperplasia, Retinal Dysplasia, and Pituitary TumorsCell, 1996
- Cell Cycle and Cancer: Critical Events at the G1 Restriction PointCritical Reviews™ in Oncogenesis, 1996
- Role of the Ubiquitin-Proteasome Pathway in Regulating Abundance of the Cyclin-Dependent Kinase Inhibitor p27Science, 1995
- Association of p53 Gene and Protein Alterations with Metastases in Colorectal CancerThe American Journal of Surgical Pathology, 1995
- p27, a novel inhibitor of G1 cyclin-Cdk protein kinase activity, is related to p21Published by Elsevier ,1994
- p27Kip1, a cyclin-Cdk inhibitor, links transforming growth factor-beta and contact inhibition to cell cycle arrest.Genes & Development, 1994
- A genetic model for colorectal tumorigenesisCell, 1990