PPARα-mediated remodeling of repolarizing voltage-gated K+ (Kv) channels in a mouse model of metabolic cardiomyopathy
- 1 June 2008
- journal article
- research article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 44 (6) , 1002-1015
- https://doi.org/10.1016/j.yjmcc.2008.03.023
Abstract
No abstract availableKeywords
Funding Information
- National Institutes of Health (P50 HL077113, R01HL-034161, R01HL-066388)
- American Heart Association
This publication has 59 references indexed in Scilit:
- The neuronal background K2P channels: focus on TREK1Nature Reviews Neuroscience, 2007
- DITPA restores the repolarizing potassium currents Itof and Iss in cardiac ventricular myocytes of diabetic ratsLife Sciences, 2006
- Pressure overload causes cardiac hypertrophy in β1-adrenergic and β2-adrenergic receptor double knockout miceJournal Of Hypertension, 2006
- Redox regulation of Ito remodeling in diabetic rat heartAmerican Journal of Physiology-Heart and Circulatory Physiology, 2005
- Selective elimination ofIK,slow1in mouse ventricular myocytes expressing a dominant negative Kv1.5α subunitAmerican Journal of Physiology-Heart and Circulatory Physiology, 2004
- Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT MethodMethods, 2001
- The consequences of disrupting cardiac inwardly rectifying K+ current (IK1) as revealed by the targeted deletion of the murine Kir2.1 and Kir2.2 genesThe Journal of Physiology, 2001
- Downregulation of K+ Channel Genes Expression in Type I Diabetic CardiomyopathyBiochemical and Biophysical Research Communications, 2001
- Simultaneous Activation of p38 MAPK and p42/44 MAPK by ATP Stimulates the K+ Current ITREK in CardiomyocytesPublished by Elsevier ,2000
- Myocardial substrate metabolism: Implications for diabetic CardiomyopathyJournal of Molecular and Cellular Cardiology, 1995