Inhibition of drug‐induced Fas ligand transcription and apoptosis by Bcl‐XL
- 1 January 2001
- journal article
- research article
- Published by Springer Nature in Molecular and Cellular Biochemistry
- Vol. 225 (1/2) , 7-20
- https://doi.org/10.1023/a:1012203110027
Abstract
Fas/Fas ligand system triggers apoptosis in many cell types. Bcl‐XL overexpresion antagonizes Fas/Fas ligand‐mediated cell death. The mechanism by which Bcl-XLinfluences Fas‐mediated cell death is...Keywords
This publication has 60 references indexed in Scilit:
- Bcl-2–Mediated Drug ResistanceThe Journal of Experimental Medicine, 1999
- Deletion of the loop region of Bcl-2 completely blocks paclitaxel-induced apoptosisProceedings of the National Academy of Sciences, 1999
- Bcl-xL Functions Downstream of Caspase-8 to Inhibit Fas- and Tumor Necrosis Factor Receptor 1-induced Apoptosis of MCF7 Breast Carcinoma CellsJournal of Biological Chemistry, 1998
- TRANSCRIPTION FACTORS OF THE NFAT FAMILY:Regulation and FunctionAnnual Review of Immunology, 1997
- Suppression of signalling through transcription factor NF-AT by interactions between calcineurin and Bcl-2Nature, 1997
- Role of kinases and the phosphatase calcineurin in the nuclear shuttling of transcription factor NF-AT4Nature, 1996
- Fas Ligand-Induced Apoptosis as a Mechanism of Immune PrivilegeScience, 1995
- FADD, a novel death domain-containing protein, interacts with the death domain of fas and initiates apoptosisCell, 1995
- Fas(CD95)/FasL interactions required for programmed cell death after T-cell activationNature, 1995
- Constitutive and induced expression of APO-1, a new member of the nerve growth factor/tumor necrosis factor receptor superfamily, in normal and neoplastic cells.1993