ERK and p38 MAP Kinase Pathways Are Mediators of Intestinal Epithelial Wound-Induced Signal Transduction
- 1 April 1997
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 233 (2) , 389-394
- https://doi.org/10.1006/bbrc.1997.6469
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Egr-1-Induced Endothelial Gene Expression: A Common Theme in Vascular InjuryScience, 1996
- Tobacco MAP Kinase: A Possible Mediator in Wound Signal Transduction PathwaysScience, 1995
- Present views on restitution of gastrointestinal epitheliumDigestive Diseases and Sciences, 1995
- How MAP Kinases Are RegulatedJournal of Biological Chemistry, 1995
- Pro-inflammatory Cytokines and Environmental Stress Cause p38 Mitogen-activated Protein Kinase Activation by Dual Phosphorylation on Tyrosine and ThreonineJournal of Biological Chemistry, 1995
- Hepatocyte Growth Factor/Scatter Factor Modulates Intestinal Epithelial Cell Proliferation and MigrationBiochemical and Biophysical Research Communications, 1994
- Fibroblast growth factors modulate intestinal epithelial cell growth and migrationGastroenterology, 1994
- Cytokine modulation of intestinal epithelial cell restitution: Central role of transforming growth factor βGastroenterology, 1993
- An early molecular component of the wound healing response in rat embryos—induction of c-fos protein in cells at the epidermal wound marginMechanisms of Development, 1992
- Epithelial Restitution in the Gastrointestinal TractJournal of Clinical Gastroenterology, 1988