Increased Adipose Tissue Expression of Hepcidin in Severe Obesity Is Independent From Diabetes and NASH
Top Cited Papers
- 1 September 2006
- journal article
- research article
- Published by Elsevier in Gastroenterology
- Vol. 131 (3) , 788-796
- https://doi.org/10.1053/j.gastro.2006.07.007
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- Synthetic hepcidin causes rapid dose-dependent hypoferremia and is concentrated in ferroportin-containing organsBlood, 2005
- Hepcidin Regulates Cellular Iron Efflux by Binding to Ferroportin and Inducing Its InternalizationScience, 2004
- Weight loss regulates inflammation‐related genes in white adipose tissue of obese subjectsThe FASEB Journal, 2004
- Increased hepcidin expression and hypoferraemia associated with an acute phase response are not affected by inactivation of HFEBritish Journal of Haematology, 2004
- Hepcidin, a putative mediator of anemia of inflammation, is a type II acute-phase proteinBlood, 2003
- A New Mouse Liver-specific Gene, Encoding a Protein Homologous to Human Antimicrobial Peptide Hepcidin, Is Overexpressed during Iron OverloadJournal of Biological Chemistry, 2001
- Hepcidin, a Urinary Antimicrobial Peptide Synthesized in the LiverJournal of Biological Chemistry, 2001
- Microtubule Integrity Regulates Src-like and Extracellular Signal-regulated Kinase Activities in Human Pro-monocytic CellsJournal of Biological Chemistry, 1998
- Subcutaneous Adipose Tissue Releases Interleukin-6, But Not Tumor Necrosis Factor- , in VivoJournal of Clinical Endocrinology & Metabolism, 1997
- HYPOFERRÆMIA IN OBESE ADOLESCENTSThe Lancet, 1962