A Cellular Model for Long QT Syndrome
Open Access
- 1 October 1997
- journal article
- Published by Elsevier
- Vol. 272 (42) , 26505-26510
- https://doi.org/10.1074/jbc.272.42.26505
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Electrostatic interactions between transmembrane segments mediate folding of Shaker K+ channel subunitsBiophysical Journal, 1997
- Potassium Channel ä and β Subunits Assemble in the Endoplasmic ReticulumPublished by Elsevier ,1997
- Voltage-gated K+ Channels Contain Multiple Intersubunit Association SitesJournal of Biological Chemistry, 1996
- Episodic ataxia results from voltage-dependent potassium channels with altered functionsNeuron, 1995
- A molecular basis for cardiac arrhythmia: HERG mutations cause long QT syndromeCell, 1995
- Assembly of mammalian voltage-gated potassium channels: Evidence for an important role of the first transmembrane segmentNeuron, 1994
- Deletion analysis of K+ channel assemblyNeuron, 1993
- Specification of Subunit Assembly by the Hydrophilic Amino-Terminal Domain of the Shaker Potassium ChannelScience, 1992
- Determination of the subunit stoichiometry of a voltage-activated potassium channelNature, 1991
- Protein Oligomerization in the Endoplasmic ReticulumAnnual Review of Cell Biology, 1989