Transcriptional regulatory networks in Th17 cell differentiation
- 1 April 2009
- journal article
- review article
- Published by Elsevier in Current Opinion in Immunology
- Vol. 21 (2) , 146-152
- https://doi.org/10.1016/j.coi.2009.03.001
Abstract
No abstract availableKeywords
This publication has 63 references indexed in Scilit:
- IRF4 is essential for IL-21-mediated induction, amplification, and stabilization of the Th17 phenotypeProceedings of the National Academy of Sciences, 2008
- IRF-4-Binding Protein Inhibits Interleukin-17 and Interleukin-21 Production by Controlling the Activity of IRF-4 Transcription FactorImmunity, 2008
- IL-6 controls Th17 immunity in vivo by inhibiting the conversion of conventional T cells into Foxp3+regulatory T cellsProceedings of the National Academy of Sciences, 2008
- Retinoic Acid Enhances Foxp3 Induction Indirectly by Relieving Inhibition from CD4+CD44hi CellsImmunity, 2008
- Interactions among the transcription factors Runx1, RORγt and Foxp3 regulate the differentiation of interleukin 17–producing T cellsNature Immunology, 2008
- Specific Microbiota Direct the Differentiation of IL-17-Producing T-Helper Cells in the Mucosa of the Small IntestineCell Host & Microbe, 2008
- TGF-β: A Master of All T Cell TradesCell, 2008
- Molecular Antagonism and Plasticity of Regulatory and Inflammatory T Cell ProgramsPublished by Elsevier ,2008
- Aryl hydrocarbon receptor regulates Stat1 activation and participates in the development of Th17 cellsProceedings of the National Academy of Sciences, 2008
- IL-21 and TGF-β are required for differentiation of human TH17 cellsNature, 2008