GLUCAGON DECREASES CYTOKINE INDUCTION OF NITRIC OXIDE SYNTHASE AND ACTION ON INSULIN SECRETION IN RIN5F CELLS AND RAT AND HUMAN ISLETS OF LANGERHANS
- 31 August 1999
- Vol. 11 (8) , 585-592
- https://doi.org/10.1006/cyto.1998.0486
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Interferon-γ Increases the Sensitivity of Islets of Langerhans for Inducible Nitric-oxide Synthase Expression Induced by Interleukin 1Journal of Biological Chemistry, 1997
- Tumor Necrosis Factor-α and Interferon-γ Inhibit Insulin Secretion and Cause DNA Damage in Unweaned-Rat Islets: Extent of Nitric Oxide InvolvementDiabetes, 1996
- The effect of nitric oxide donors on insulin secretion, cyclic GMP and cyclic AMP in rat islets of Langerhans and the insulin-secreting cell lines HIT-T15 and RINm5FMolecular and Cellular Endocrinology, 1994
- Effects of cytokines and nitric oxide donors on insulin secretion, cyclic GMP and DNA damage: relation to nitric oxide productionBiochemical Society Transactions, 1994
- Nicotinamide and dexamethasone inhibit interleukin-1-induced nitric oxide production by RINm5F cells without decreasing messenger ribonucleic acid expression for nitric oxide synthase.Endocrinology, 1993
- Interleukin-1? effects on cyclic GMP and cyclic AMP in cultured rat islets of Langerhans ? arginine ? dependence and relationship to insulin secretionDiabetologia, 1993
- Interleukin‐1β induces the expression of an isoform of nitric oxide synthase in insulin‐producing cells, which is similar to that observed in activated macrophagesFEBS Letters, 1992
- Interleukin-lβ-Induced Nitric Oxide Production in Isolated Rat Pancreatic Islets Requires Gene Transcription and May Lead to Inhibition of the Krebs Cycle Enzyme Aconitase*Endocrinology, 1991
- Inhibition of insulin secretion by interleukin‐1β and tumour necrosis factor‐α via an L‐arginine‐dependent nitric oxide generating mechanismFEBS Letters, 1990
- Metabolism and β-cell function of rat pancreatic islets exposed to human interleukin-1β in the presence of a high glucose concentrationImmunology Letters, 1990