Much progress has been made in the study of sarcoid since Boeck1revealed this clinical entity. In addition to Boeck2dermatologists are indebted to Jüngling,3Schaumann,4Kuznitzky and Bittorf,5Kyrle,6Jadassohn7and many others for the attainment of a sound understanding of this disease. It has been shown repeatedly8that the cutaneous eruption is merely a visible indication of a systemic disease, the tubercle bacillus being the most likely inciting agent. Kyrle's6bsuccess in demonstrating the presence of the tubercle bacillus in the early efflorescences in the banal inflammatory response and its gradual disappearance simultaneous with the onset of a tuberculoid histologic structure in mature lesions is undeniable evidence of tuberculous causation. These observations were confirmed by Ruete.9It thus became evident that the local immunologic response was capable of altering or destroying the micro-organism and hence preventing its detection