Where does damage lead to enhanced food aversion: the ventral pallidum/substantia innominata or lateral hypothalamus?
- 1 October 1993
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 624 (1-2) , 1-10
- https://doi.org/10.1016/0006-8993(93)90053-p
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Motivational-sensorimotor interaction controls aphagia and exaggerated treading after striatopallidal lesions.Behavioral Neuroscience, 1990
- The quinolinic acid model of Huntington's disease: Locomotor abnormalitiesExperimental Neurology, 1989
- A triggered hyperkinesia induced in rats by lesions of the corpus striatumExperimental Neurology, 1988
- Lesions of the globus pallidus and striatum attenuate ketocyclazocine-induced feedingPhysiology & Behavior, 1984
- An analysis of feeding and sensorimotor abilities of rats after decortication.Journal of Comparative and Physiological Psychology, 1981
- Aphagia and Adipsia After Preferential Destruction of Nerve Cell Bodies in HypothalamusScience, 1978
- Two types of aphagia and two types of sensorimotor impairment after lateral hypothalamic lesions: Observations in normal weight, dieted, and fattened rats.Journal of Comparative and Physiological Psychology, 1978
- Projections from the amygdaloid complex to the cerebral cortex and thalamus in the rat and catJournal of Comparative Neurology, 1977
- Sensorimotor functions of the striatopallidal system and lateral hypothalamus and consummatory behavior in rats.Journal of Comparative and Physiological Psychology, 1973
- Recovery from the Failure to Eat Produced by Hypothalamic LesionsScience, 1954